Da: GreatBookPrices, Columbia, MD, U.S.A.
Condizione: New.
Da: Lucky's Textbooks, Dallas, TX, U.S.A.
EUR 101,23
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New.
Da: Ria Christie Collections, Uxbridge, Regno Unito
EUR 112,13
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. In.
Da: GreatBookPricesUK, Woodford Green, Regno Unito
EUR 113,30
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New.
Condizione: New. pp. 288.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, 2012
ISBN 10: 3642741819 ISBN 13: 9783642741814
Da: Revaluation Books, Exeter, Regno Unito
EUR 148,91
Quantità: 2 disponibili
Aggiungi al carrelloPaperback. Condizione: Brand New. reprint edition. 284 pages. 9.60x6.70x0.70 inches. In Stock.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, 2011
ISBN 10: 3642741819 ISBN 13: 9783642741814
Da: AHA-BUCH GmbH, Einbeck, Germania
EUR 106,99
Quantità: 1 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Sixty years ago, G. Fanconi published a paper entitled: 'Familiiire infantile pemiziosaartige Aniimie (pemizioses Blutbild und Konstitu tion)', in which he reported that this type of severe aplastic anemia represents a hereditary disease distinct from other pancytopenias of childhood (Fanconi 1927). Later this syndrome was named Fan coni anemia (FA; van Leeuwen 1933). A more recent study of the genetics of FA confirmed that the syndrome is inherited in an au tosomal recessive manner (Schroeder et al. 1976). Prenatal diagno sis in FA families showed that about 25% of fetuses are affected (Auerbach et al. 1985, 1986). In 1964, Schroeder et al. discovered high frequencies of chro mosomal aberrations in cultured peripheral blood lymphocytes from patients with FA. Schuler et al. (1969) reported that cells from FA patients are particularly sensitive to the chromosome-breaking activity or clastogenic effect of a polyfunctional alkylating agent. Since that time, studies of baseline and induced frequencies of chromosomal aberrations have been used for the identification of patients with FA. There is now a large body of data concerning the possible mechanism(s) underlying the hypersensitivity of FA cells to DNA cross-linking agents, the biochemical basis for which is still unknown. Complementation analysis, using cells from different FA pa tients, has demonstrated genetic heterogeneity in the syndrome.
Lingua: Inglese
Editore: Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 1989
ISBN 10: 354050401X ISBN 13: 9783540504016
Da: Mispah books, Redhill, SURRE, Regno Unito
EUR 171,23
Quantità: 1 disponibili
Aggiungi al carrelloHardcover. Condizione: Very Good. Very GoodDust Jacket may NOT BE INCLUDED.CDs may be missing. book.
Da: GreatBookPricesUK, Woodford Green, Regno Unito
EUR 193,81
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: As New. Unread book in perfect condition.
Da: Mispah books, Redhill, SURRE, Regno Unito
EUR 184,31
Quantità: 1 disponibili
Aggiungi al carrelloPaperback. Condizione: Like New. Like New. book.
Da: GreatBookPrices, Columbia, MD, U.S.A.
Condizione: As New. Unread book in perfect condition.
Lingua: Tedesco
Editore: Springer Berlin Heidelberg 1989-01-01, 1989
ISBN 10: 3642739385 ISBN 13: 9783642739385
Da: Chiron Media, Wallingford, Regno Unito
EUR 59,19
Quantità: 10 disponibili
Aggiungi al carrelloPaperback. Condizione: New.
Lingua: Inglese
Editore: Springer Berlin Heidelberg Dez 2011, 2011
ISBN 10: 3642741819 ISBN 13: 9783642741814
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germania
EUR 96,29
Quantità: 2 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Sixty years ago, G. Fanconi published a paper entitled: 'Familiiire infantile pemiziosaartige Aniimie (pemizioses Blutbild und Konstitu tion)', in which he reported that this type of severe aplastic anemia represents a hereditary disease distinct from other pancytopenias of childhood (Fanconi 1927). Later this syndrome was named Fan coni anemia (FA; van Leeuwen 1933). A more recent study of the genetics of FA confirmed that the syndrome is inherited in an au tosomal recessive manner (Schroeder et al. 1976). Prenatal diagno sis in FA families showed that about 25% of fetuses are affected (Auerbach et al. 1985, 1986). In 1964, Schroeder et al. discovered high frequencies of chro mosomal aberrations in cultured peripheral blood lymphocytes from patients with FA. Schuler et al. (1969) reported that cells from FA patients are particularly sensitive to the chromosome-breaking activity or clastogenic effect of a polyfunctional alkylating agent. Since that time, studies of baseline and induced frequencies of chromosomal aberrations have been used for the identification of patients with FA. There is now a large body of data concerning the possible mechanism(s) underlying the hypersensitivity of FA cells to DNA cross-linking agents, the biochemical basis for which is still unknown. Complementation analysis, using cells from different FA pa tients, has demonstrated genetic heterogeneity in the syndrome. 288 pp. Englisch.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, 2011
ISBN 10: 3642741819 ISBN 13: 9783642741814
Da: moluna, Greven, Germania
EUR 92,27
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. I Clinical and Therapeutical Aspects.- International Fanconi Anemia Registry: First Report.- Fanconi Anemia in The Netherlands.- Clinical Aspects of a Cluster of 42 Patients in South Africa with Fanconi Anemia.- Therapeutic Aspects of Fanconi Anemia.- Bone .
Da: Majestic Books, Hounslow, Regno Unito
EUR 142,34
Quantità: 4 disponibili
Aggiungi al carrelloCondizione: New. Print on Demand pp. 288 Figure, Illus.
Da: Biblios, Frankfurt am main, HESSE, Germania
EUR 147,26
Quantità: 4 disponibili
Aggiungi al carrelloCondizione: New. PRINT ON DEMAND pp. 288.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, Springer Berlin Heidelberg Dez 2011, 2011
ISBN 10: 3642741819 ISBN 13: 9783642741814
Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germania
EUR 106,99
Quantità: 1 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Sixty years ago, G. Fanconi published a paper entitled: 'Familiiire infantile pemiziosaartige Aniimie (pemizioses Blutbild und Konstitu tion)', in which he reported that this type of severe aplastic anemia represents a hereditary disease distinct from other pancytopenias of childhood (Fanconi 1927). Later this syndrome was named Fan coni anemia (FA; van Leeuwen 1933). A more recent study of the genetics of FA confirmed that the syndrome is inherited in an au tosomal recessive manner (Schroeder et al. 1976). Prenatal diagno sis in FA families showed that about 25% of fetuses are affected (Auerbach et al. 1985, 1986). In 1964, Schroeder et al. discovered high frequencies of chro mosomal aberrations in cultured peripheral blood lymphocytes from patients with FA. Schuler et al. (1969) reported that cells from FA patients are particularly sensitive to the chromosome-breaking activity or clastogenic effect of a polyfunctional alkylating agent. Since that time, studies of baseline and induced frequencies of chromosomal aberrations have been used for the identification of patients with FA. There is now a large body of data concerning the possible mechanism(s) underlying the hypersensitivity of FA cells to DNA cross-linking agents, the biochemical basis for which is still unknown. Complementation analysis, using cells from different FA pa tients, has demonstrated genetic heterogeneity in the syndrome.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 288 pp. Englisch.
Da: preigu, Osnabrück, Germania
EUR 95,70
Quantità: 5 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. Fanconi Anemia | Clinical, Cytogenetic and Experimental Aspects | Traute M. Schroeder-Kurth (u. a.) | Taschenbuch | xx | Englisch | 2011 | Springer | EAN 9783642741814 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu Print on Demand.