Benlian pascale (33 risultati)

Couverture rigide. Condizione: Bon. Médecine. XI + 253 p. Ces anomalies très fréquentes du métabolisme des lipoprotéines qui sont les dyslipoprotéinémies représentent l'un des problèmes majeurs de santé publique dans notre pays car elles prédisposent aux maladies cardiovasculaires. En outre, elles regroupent un ensemble complexe de maladies polyfactorielles, dans lequel facteurs d'environnement et facteurs génétiques interagissent à des degrés divers suivant les individus. L'essor des techniques de biologie moléculaire a permis des avancées importantes dans la compréhension des mécanismes génétiques qui sous-tendent ces pathologies, par une mesure directe du génotype, sur le génome humain et leur rôle dans la pathogenèse de nombreuses dyslipoprotéinémies est maintenant bien connu. Cet ouvrage propose une synthèse articulant des connaissances cliniques, biochimiques et celles, plus nouvelles, de la génétiques moléculaire dans ce domaine. Il présente des revues générales très détaillées, d'une part, sur la physiologie et le métabolisme des lipoprotéines et, d'autre part, sur l'approche dite du (des) gène(s)-candidat(s), son principe, ses possibilités et ses limites. L'état d'avancée de cette approche dans le domaine des dyslipoprotéinémies, en relation avec l'arthérosclérose coronarienne, est ensuite présenté de façon exhaustive. L'ensemble constitue un ouvrage très didactiques, dans lequel l'auteur discute en profondeur tous les aspects conceptueles se rapportant à l'approche "gène-candidat" dans des études d'épidémiologie génétique, en replaçant cette approche dans l'évolution des méthodes employées pour l'étude des hyperlipidémies. 17x25 cm. 253 p. Cartonnage éditeur. Bon état. Intérieur en bon état. Nom de l'ancien propriétaire en page de titre (cachBerger-Levrault et Cie Nancy. in-4°.…

hardcover. Condizione: Good. Connecting readers with great books since 1972! Used textbooks may not include companion materials such as access codes, etc. May have some wear or writing/highlighting. We ship orders daily and Customer Service is our top priority.

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HRD. Condizione: New. New Book. Shipped from UK. Established seller since 2000.

HRD. Condizione: New. New Book. Shipped from UK. Established seller since 2000.

Condizione: New.

Condizione: As New. Unread book in perfect condition.

Condizione: NEW.

Condizione: As New. Unread book in perfect condition.

Condizione: New. In English.

Hardback. Condizione: New. Profound mortality rates, due to cardiovascular disease, are a worldwide problem. One of the consequences from complications of this silent and chronic arterial disease is atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, disorders of the metabolism of soluble transporter of lipids in extracellular spaces of the human body (including blood), called lipoproteins, are major cardiovascular risk factors and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than 50 genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of these observations has refined our knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system.These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations. The book is a fitting addition to the Endocrine Updates series. The aim of these publications is to provide the clinician with cutting edge, yet succinct, access to the latest advances in endocrinology.…

Condizione: New.

Condizione: New. In English.

Hardback. Condizione: New. New copy - Usually dispatched within 4 working days.

Condizione: New. Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and t.

Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations.…

Condizione: New. pp. 332.

Condizione: New. The study of genetic factors has taken a different path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism are identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. Series: Basic Science for the Cardiologist. Num Pages: 315 pages, biography. BIC Classification: MFGM; MFN; MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 234 x 156 x 19. Weight in Grams: 1420. . 2001. Hardback. . . . .…

Condizione: New. The study of genetic factors has taken a different path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism are identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. Series: Basic Science for the Cardiologist. Num Pages: 315 pages, biography. BIC Classification: MFGM; MFN; MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 234 x 156 x 19. Weight in Grams: 1420. . 2001. Hardback. . . . . Books ship from the US and Ireland.…

Hardback. Condizione: New. Profound mortality rates, due to cardiovascular disease, are a worldwide problem. One of the consequences from complications of this silent and chronic arterial disease is atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, disorders of the metabolism of soluble transporter of lipids in extracellular spaces of the human body (including blood), called lipoproteins, are major cardiovascular risk factors and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than 50 genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of these observations has refined our knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system.These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations. The book is a fitting addition to the Endocrine Updates series. The aim of these publications is to provide the clinician with cutting edge, yet succinct, access to the latest advances in endocrinology.…

Paperback. Condizione: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

Condizione: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

Buch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations. 332 pp. Englisch.…

Hardcover. Condizione: Brand New. 315 pages. 9.25x5.75x0.75 inches. In Stock. This item is printed on demand.

Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and t. …

Buch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease. Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors. The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations.…

Buch. Condizione: Neu. Genetics of Dyslipidemia | Pascale Benlian | Buch | xi | Englisch | 2001 | Springer US | EAN 9780792373629 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu Print on Demand.…

Taschenbuch. Condizione: Neu. Genetics of Dyslipidemia | Pascale Benlian | Taschenbuch | xi | Englisch | 2012 | Springer US | EAN 9781461355939 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu Print on Demand.…

Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease.Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors.The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 332 pp. Englisch.…

Buch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Profound mortality rates, due to cardiovascular disease, are a worldwide problem. Cardiovascular disease results from complications of a silent and chronic arterial disease: atherosclerosis. The challenge for the practitioner is adapting diagnostic and therapeutic responses to prevent this common and complex disease.Dyslipidemia, are disorders of the metabolism of soluble transporters of lipids in extracellular spaces of the human body (including blood), called lipoproteins. They are major cardiovascular risk factors, causally related with atherosclerosis and are themselves multifactorial diseases, resulting from interactions between genetic and environmental factors.The study of genetic factors has recently taken a new path with the study of DNA as an experimental object. More than fifty genes of lipoprotein metabolism have been identified in both their physiological actions and their contribution to the pathogenesis of human dyslipidemia. The diversity of observations has refined our current knowledge of the control of lipid metabolism and energy homeostasis in living organisms beyond the limits of the cardiovascular system (e.g., brain, immune system, and development). These studies have given way to a shake-up of former phenotypic classifications, distinguishing new entities, defining targeted therapeutic strategies, providing a basis for different patterns of disease distribution in human populations.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 332 pp. Englisch.…