Dennis selkoe (67 risultati)

Hardcover/Pappeinband. Condizione: Gut. Auflage: 1. 240 Seiten Sauberes, gepflegtes Exemplar. Am oberen Kap. leicht berieben. F208 Sprache: Englisch Gewicht in Gramm: 554.

Condizione: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.

Condizione: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.

Condizione: Good. Your purchase helps support Sri Lankan Children's Charity 'The Rainbow Centre'. Ex-library, so some stamps and wear, but in good overall condition. Our donations to The Rainbow Centre have helped provide an education and a safe haven to hundreds of children who live in appalling conditions.…

Hardcover. Condizione: Fine. Xvii, 180 Pp. Brown Cloth, Gilt. Ownership Signature Of Prominent Cardiac Surgeon Richard J. Bing, Listed In The Book As A Member Of The Harvey Society Of New York.

Hardcover. Condizione: New. 1st Edition. New book, never read. Pages clean and crisp, spine unbroken. Oversized book, may incur additional shipping charges. 1025A.

Condizione: Used. pp. xix + 243.

Condizione: Used. pp. xix + 243 60 Figures.

Condizione: New. In English.

Condizione: Used. pp. xix + 243.

Condizione: New. In English.

Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Promising effects in mice of immunization by ß-amyloid stimulated substantial research efforts and high hopes. In retrospect, this study appears simultaneously logical, consistent as it is with the amyloid theory central today in the field of Alzheimer's disease studies, and paradoxical, because it involved using the toxic substance itself for a treatment benefit. The research thus begun opens up multiple perspectives for experimentation and for treatment. Clinical trials began, but had to be stopped in January 2002 because of serious side effects. The editors' objective for this book is not to arouse false hopes for a therapeutic project that must still be proven, but to discuss the availableinformation and the questions it raises.…

Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Like the unflinching gaze of Captain Ahab walking the deck of the Pequod, Alzheimer researchers have had their sights fixed firmly on the disease for many years. Now, as this volume amply demonstrates, accomplished researchers from other fields, who have thought deeply about cell biological problems are applying their insights to Alzheimer's disease. The contri butions here represent the text versions of the proceedings from the tenth 'Colloque medecine et recherche' of the Fondation IPSEN devoted to research on Alzheimer's disease. The symposium, entitled 'Alzheimer's Disease: Lessons from Cell Biology' was held in Paris on April 25, 1994. As is apparent from the varied backgrounds of the contributors, the scientific pursuit of Alzheimer's disease has begun to meld with more basic disciplines, particularly cell biology. While on the one hand, new areas of specialization are continuously emerging, the boundaries of older disciplines are increas ingly blurred. Perhaps for most of the years since the first descriptions of the disease in 1907, the science of Alzheimer's disease was descriptive, and lay in the province of pathologists. This time period, during which a great deal was learned about the topography of senile plaques and neurofibrillary tangles, culminated with an ultrastructural description of these hallmark structures. The modern era of Alzheimer's disease research opened with the iden tification of the component proteins in plaques and tangles.…

Condizione: As New. Unread book in perfect condition.

Condizione: As New. Unread book in perfect condition.

Gebunden. Condizione: New. KlappentextrnrnAlzheimer Disease represents an important area of research in neurobiology, cell biology, developmental biology, and pathology. Understanding the nature of the changes that occur in neurons as the disease progresses accumulation o.

Condizione: New.

Condizione: New. pp. xiv + 126.

Paperback. Condizione: Brand New. reprint edition. 138 pages. 9.00x6.10x0.40 inches. In Stock.

Paperback. Condizione: Brand New. reprint edition. 262 pages. 8.98x6.06x0.55 inches. In Stock.

Taschenbuch. Condizione: Neu. Alzheimer's Disease: Lessons from Cell Biology | Ken S. Kosik (u. a.) | Taschenbuch | Research and Perspectives in Alzheimer's Disease | xix | Englisch | 2011 | J.B. Metzler | EAN 9783642794254 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu. …

Condizione: New.

Condizione: As New. Unread book in perfect condition.

Hardcover. Condizione: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

Hardback. Condizione: New.

Condizione: New.

Condizione: Sehr gut. Zustand: Sehr gut | Sprache: Englisch | Produktart: Bücher | During most of the 20th century, neurodegenerative diseases remained among the most enigmatic disorders of medicine. The scientific study of these conditions was descriptive in nature, detailing the clinical and neuropathological phenotypes associated with various diseases, but etiologies and pathogenic mechanisms remained obscure. Beginning in the 1970s, advances in two principal areas ¿ biochemical pathology and molecular genetics ¿ combined to yield powerful clues to the molecular underpinnings of several previously "idiopathic" brain disorders. Among the classical neurodegenerative diseases, perhaps the most rapid progress occurred in research on Alzheimer¿s disease (AD). In disorders like Huntington¿s disease, amyotrophic lateral sclerosis and even Parkinson¿s disease, unbiased genetic screens, linkage analysis and positional cloning have identified causative genes that subsequently allowed the formulation of specific biochemical hypotheses. In sharp contrast, modern research on AD developed in the opposite order: the identification of the protein subunits of the classical brain lesions guided geneticists to disease-inducing genes, for example, APP, apolipoprotein E and tau. Thus, a biochemical hypothesis of disease - that AD is a progressive cerebral amyloidosis caused by the aggregation of the amyloid b-protein (Ab) - preceded and enabled the discovery of etiologies.…

Condizione: New.

Gebunden. Condizione: New.

Condizione: New.