Reulen philippon (15 risultati)
Lingua: Inglese
Editore: Springer 1989 1989
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Antiquariat Artemis Lorenz & Lorenz GbR, Leipzig, , GermaniaAntiquariat Artemis Lorenz & Lorenz GbR
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* Gebundene Ausgabe * Verlag: Springer-Verlag (März 1989) * Sprache: Englisch * ISBN-10: 0387820965 * ISBN-13: 978-0387820965 The papers presented in this supplement volume of "Acta Neurochirugica" cover a broad spectrum of topics, from basic data on the pathophysiology of Subarachnoid Haemorrhage (SAH) and delayed ischaemic dys…function to the clinical use of Nimodipine which has been largely documented among calcium inhibitors for its cerebro-vascular properties. The first two presentations deal with the pathophysiological events following SAH, and discuss a variety of mechanisms which may be responsible for the neurological dysfunction. The second part discusses the cerebro-vascular and direct cerebral effects of Nimodipine and its evaluation by transcranial Doppler sonography and the interactions of this with general anaesthesia. Therapeutic aspects constitute the third part of this volume. On the basis of present knowledge and experience, it seems that early surgery combined with prophylactic treatment represents the best option for patients after aneurysmal rupture gutes Exemplar, Einband mit kleinen altersbedingten Spuren * Verlag: Springer 1989.
Lingua: Inglese
Editore: Springer 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: GreatBookPrices, Columbia, MD, U.S.A.GreatBookPrices
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Lingua: Inglese
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Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Ria Christie Collections, Uxbridge, Regno UnitoRia Christie Collections
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Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Zubal-Books, Since 1961, Cleveland, OH, U.S.A.Zubal-Books, Since 1961
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Condizione: Good. 55 pp., Hardcover, ex library, else text clean and binding tight. - If you are reading this, this item is actually (physically) in our stock and ready for shipment once ordered. We are not bookjackers. Buyer is responsible for any additional duties, taxes, or fees required by recipient's country.
Lingua: Inglese
Editore: Springer 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Books Puddle, New York, NY, U.S.A.Books Puddle
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Condizione: New. pp. 68.
Lingua: Inglese
Editore: Springer Vienna 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Revaluation Books, Exeter, , Regno UnitoRevaluation Books
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Paperback. Condizione: Brand New. 62 pages. 10.98x8.27x0.16 inches. In Stock.
Lingua: Inglese
Editore: Springer, Springer 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: AHA-BUCH GmbH, Einbeck, GermaniaAHA-BUCH GmbH
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Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by its title 'Prevention and treatment of del…ayed ischaemic dysfunction in patients with subarachnoid haemorrhage: an update', papers presented at the Symposium covered a broad spectrum from some basic data on pathophysiology of subarachnoid haemorrhage (SAH) and delayed ischaemic dysfunction, to the clinical use of nimodipine, which has been largely documented among calcium inhibitors for its cerebrovascular properties. This supplementum of Acta Neurochirurgica is based upon the papers presented. Some articles, however, have been extended to take into account further results in order to present a broad view of the subject. The Symposium started with two presentations concerning general aspects of SAH: in the first one, B. Voldby reviewed the pathophysiological events following SAH: if modifications of intracranial pressure (ICP) and decrease of cerebral blood flow (CBF) are the primary alterations, metabolic changes (particularly reduction in CMR0 ) 2 contribute rapidly to disturbances of brain circulation. A variety of mechanisms may be responsible for the neurological dysfunction: most of them are directly related to the reduction in cerebral perfusion pressure (CPP) which may result in ischaemia. This fall in CPP (either due to elevation of ICP or reduction of mean arterial blood pressure) is observed under different pathophysiological conditions, discussed in the paper by A. D. Mendelow.
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Da: Antiq. F.-D. Söhn - Medicusbooks.Com, Marburg, , GermaniaAntiq. F.-D. Söhn - Medicusbooks.Com
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Acta Neurochir., Suppl., 45. - Wien, New York, 1988, 4°, (6), 55, (5) pp., 16 Abbildungen, orig. Leinenband. First Edition! "During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by…its title "Prevention and treatment of delayed ischaemic dysfunction in patients with subarachnoid haemorrhage: an update", papers presented at the Symposium covered a broad spectrum from some basic data on pathophysiology of subarachnoid haemorrhage (SAH) and delayed ischaemic dysfunction, to the clinical use of nimodipine, which has been largely documented among calcium inhibitors for its cerebrovascular properties. This supplementum of Acta Neurochirurgica is based upon the papers presented. Some articles, however, have been extended to take into account further results in order to present a broad view of the subject. The Symposium started with two presentations concerning general aspects of SAH: in the first one, B. Voldby reviewed the pathophysiological events following SAH: if modifications of intracranial pressure (ICP) and decrease of cerebral blood flow (CBF) are the primary alterations, metabolic changes (particularly reduction in CMR0 ) 2 contribute rapidly to disturbances of brain circulation. A variety of mechanisms may be responsible for the neurological dysfunction: most of them are directly related to the reduction in cerebral perfusion pressure (CPP) which may result in ischaemia. This fall in CPP (either due to elevation of ICP or reduction of mean arterial blood pressure) is observed under different pathophysiological conditions, discussed in the paper by A. D. Mendelow." Springer-Verlag.
Lingua: Inglese
Editore: Springer, Springer Dez 1988 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, , GermaniaBuchWeltWeit Ludwig Meier e.K.
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Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by its title 'Prevention and…treatment of delayed ischaemic dysfunction in patients with subarachnoid haemorrhage: an update', papers presented at the Symposium covered a broad spectrum from some basic data on pathophysiology of subarachnoid haemorrhage (SAH) and delayed ischaemic dysfunction, to the clinical use of nimodipine, which has been largely documented among calcium inhibitors for its cerebrovascular properties. This supplementum of Acta Neurochirurgica is based upon the papers presented. Some articles, however, have been extended to take into account further results in order to present a broad view of the subject. The Symposium started with two presentations concerning general aspects of SAH: in the first one, B. Voldby reviewed the pathophysiological events following SAH: if modifications of intracranial pressure (ICP) and decrease of cerebral blood flow (CBF) are the primary alterations, metabolic changes (particularly reduction in CMR0 ) 2 contribute rapidly to disturbances of brain circulation. A variety of mechanisms may be responsible for the neurological dysfunction: most of them are directly related to the reduction in cerebral perfusion pressure (CPP) which may result in ischaemia. This fall in CPP (either due to elevation of ICP or reduction of mean arterial blood pressure) is observed under different pathophysiological conditions, discussed in the paper by A. D. Mendelow. 68 pp. Englisch.
Lingua: Inglese
Editore: Springer 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Majestic Books, Hounslow, , Regno UnitoMajestic Books
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Condizione: New. Print on Demand pp. 68 6:B&W 8.25 x 11 in or 280 x 210 mm Perfect Bound on White w/Gloss Lam.
Lingua: Inglese
Editore: Springer 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: Biblios, frankfurt am main, HESSE, GermaniaBiblios
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Condizione: New. PRINT ON DEMAND pp. 68.
Lingua: Inglese
Editore: Springer Vienna 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: moluna, Greven, , Germaniamoluna
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Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by its title P…revention and treatment of delayed ischa.
Lingua: Inglese
Editore: Springer, Springer Dez 1988 1988
Serie: Acta Neurochirurgica Supplement, Libro 38 di 91. Libro 38 di 91 - Acta Neurochirurgica Supplement
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Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germaniabuchversandmimpf2000
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Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -During the last meeting of the European Association of Neurosurgical Societies (Barcelona 7-16 September 1987), a Symposium was devoted to the use of calcium antagonists in cerebral vasospasm. As shown by its title 'Prevention and trea…tment of delayed ischaemic dysfunction in patients with subarachnoid haemorrhage: an update', papers presented at the Symposium covered a broad spectrum from some basic data on pathophysiology of subarachnoid haemorrhage (SAH) and delayed ischaemic dysfunction, to the clinical use of nimodipine, which has been largely documented among calcium inhibitors for its cerebrovascular properties. This supplementum of Acta Neurochirurgica is based upon the papers presented. Some articles, however, have been extended to take into account further results in order to present a broad view of the subject. The Symposium started with two presentations concerning general aspects of SAH: in the first one, B. Voldby reviewed the pathophysiological events following SAH: if modifications of intracranial pressure (ICP) and decrease of cerebral blood flow (CBF) are the primary alterations, metabolic changes (particularly reduction in CMR0 ) 2 contribute rapidly to disturbances of brain circulation. A variety of mechanisms may be responsible for the neurological dysfunction: most of them are directly related to the reduction in cerebral perfusion pressure (CPP) which may result in ischaemia. This fall in CPP (either due to elevation of ICP or reduction of mean arterial blood pressure) is observed under different pathophysiological conditions, discussed in the paper by A. D. Mendelow.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 68 pp. Englisch.
