Advances in experimental medicine and biology - 9783319891033 - polyglutamine disorders: 1049 (11 risultati)
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
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Da: Ria Christie Collections, Uxbridge, Regno UnitoRia Christie Collections
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Condizione: New. In.
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
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Da: Books Puddle, New York, NY, U.S.A.Books Puddle
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EUR 165,25
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Condizione: New. pp. 469.
Lingua: Inglese
Editore: Springer International Publishing, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
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Da: moluna, Greven, Germaniamoluna
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Condizione: New.
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
Da: AHA-BUCH GmbH, Einbeck, GermaniaAHA-BUCH GmbH
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Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - This book provides a cutting-edge review of polyglutamine disorders. It primarily focuses on two main aspects: (1) the mechanisms underlying the pathologies' development and progression, and (2) the therapeutic strategies that are currently being…explored to stop or delay disease progression. Polyglutamine (polyQ) disorders are a group of inherited neurodegenerative diseases with a fatal outcome that are caused by an abnormal expansion of a coding trinucleotide repeat (CAG), which is then translated in an abnormal protein with an elongated glutamine tract (Q). To date, nine polyQ disorders have been identified and described: dentatorubral-pallidoluysian atrophy (DRPLA); Huntington's disease (HD); spinal-bulbar muscular atrophy (SBMA); and six spinocerebellar ataxias (SCA 1, 2, 3, 6, 7, and 17).The genetic basis of polyQ disorders is well established and described, and despite important advances that have opened up the possibility of generating genetic models of the disease, the mechanisms that cause neuronal degeneration are still largely unknown and there is currently no treatment available for these disorders. Further, it is believed that the different polyQ may share some mechanisms and pathways contributing to neurodegeneration and disease progression.
Altre immaginiLingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
Da: preigu, Osnabrück, Germaniapreigu
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EUR 122,20
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Taschenbuch. Condizione: Neu. Polyglutamine Disorders | Clévio Nóbrega (u. a.) | Taschenbuch | Advances in Experimental Medicine and Biology | viii | Englisch | 2019 | Springer | EAN 9783319891033 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]…com | Anbieter: preigu.
Lingua: Inglese
Editore: Springer Verlag, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
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Da: Revaluation Books, Exeter, Regno UnitoRevaluation Books
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EUR 195,44
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Paperback. Condizione: Brand New. reprint edition. 480 pages. 9.25x6.10x0.94 inches. In Stock.
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
- Print on Demand
Da: Brook Bookstore On Demand, Napoli, NA, ItaliaBrook Bookstore On Demand
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Condizione: new. Questo è un articolo print on demand.
Lingua: Inglese
Editore: Springer International Publishing Jun 2019, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
- Print on Demand
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermaniaBuchWeltWeit Ludwig Meier e.K.
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Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -This book provides a cutting-edge review of polyglutamine disorders. It primarily focuses on two main aspects: (1) the mechanisms underlying the pathologies' development and progression, and (2) the therapeutic strategies that are…currently being explored to stop or delay disease progression. Polyglutamine (polyQ) disorders are a group of inherited neurodegenerative diseases with a fatal outcome that are caused by an abnormal expansion of a coding trinucleotide repeat (CAG), which is then translated in an abnormal protein with an elongated glutamine tract (Q). To date, nine polyQ disorders have been identified and described: dentatorubral-pallidoluysian atrophy (DRPLA); Huntington's disease (HD); spinal-bulbar muscular atrophy (SBMA); and six spinocerebellar ataxias (SCA 1, 2, 3, 6, 7, and 17).The genetic basis of polyQ disorders is well established and described, and despite important advances that have opened up the possibility of generating genetic models of the disease, the mechanisms that cause neuronal degeneration are still largely unknown and there is currently no treatment available for these disorders. Further, it is believed that the different polyQ may share some mechanisms and pathways contributing to neurodegeneration and disease progression. 480 pp. Englisch.
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
- Print on Demand
Da: Majestic Books, Hounslow, Regno UnitoMajestic Books
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EUR 169,39
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Condizione: New. Print on Demand pp. 469.
Lingua: Inglese
Editore: Springer, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
- Print on Demand
Da: Biblios, frankfurt am main, HESSE, GermaniaBiblios
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EUR 173,60
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Condizione: New. PRINT ON DEMAND pp. 469.
Lingua: Inglese
Editore: Springer, Springer Jun 2019, 2019
Serie: Libro 345 di 544 - Advances in Experimental Medicine and Biology
- Brossura
- Print on Demand
Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germaniabuchversandmimpf2000
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EUR 139,09
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Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -This book provides a cutting-edge review of polyglutamine disorders. It primarily focuses on two main aspects: (1) the mechanisms underlying the pathologies¿ development and progression, and (2) the therapeutic strategies that are curr…ently being explored to stop or delay disease progression.Polyglutamine (polyQ) disorders are a group of inherited neurodegenerative diseases with a fatal outcome that are caused by an abnormal expansion of a coding trinucleotide repeat (CAG), which is then translated in an abnormal protein with an elongated glutamine tract (Q). To date, nine polyQ disorders have been identified and described: dentatorubral-pallidoluysian atrophy (DRPLA); Huntington¿s disease (HD); spinal¿bulbar muscular atrophy (SBMA); and six spinocerebellar ataxias (SCA 1, 2, 3, 6, 7, and 17).The genetic basis of polyQ disorders is well established and described, and despite important advances that have opened up the possibility of generating genetic models of the disease, the mechanisms that cause neuronal degeneration are still largely unknown and there is currently no treatment available for these disorders. Further, it is believed that the different polyQ may share some mechanisms and pathways contributing to neurodegeneration and disease progression.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 480 pp. Englisch.
