Hard Cover. Condizione: Acceptable. No Jacket. Ex-library with the usual features. The interior is clean and tight. Binding is good. Cover shows light wear. 154 pages. Ex-Library.
Lingua: Inglese
Editore: Springer-Verlag Berlin and Heidelberg GmbH & Co. K, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: JLG_livres anciens et modernes, Saint Maur des Fossés, Francia
EUR 42,07
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Très bon. 2001 ed. Nos envois se font avec suivi, pour tout problème n'hésitez pas à nous contacter pour trouver une solution.
Lingua: Inglese
Editore: Berlin ; Heidelberg ; New York ; Barcelona ; Hong Kong ; London ; Milan ; Paris ; Tokyo : Springer, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: Versandhandel Rosemarie Wassmann, Oyten, Germania
EUR 15,80
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Sehr gut. XI, 154 S. : Ill., graph. Darst. ; 24 cm Leichte Lagerspuren am Pappband sonst sehr gut. F3 Sprache: Englisch Gewicht in Gramm: 501 8° , Pp., Hardcover/Pappeinband.
Da: Ria Christie Collections, Uxbridge, Regno Unito
EUR 164,59
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. In.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, Springer Berlin Heidelberg, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: AHA-BUCH GmbH, Einbeck, Germania
EUR 160,49
Quantità: 1 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Four chapters represent the intense current effort to understand the way in which the mitochondrion controls the activation of the final stages of cell death. Another four articles attack the problem from the other side. How do specific insults in particular human or mouse neuro-degenerative diseases translate into mechanisms that will not only allow us to better understand what is happening in these patients but also, with luck, allow for development of more efficient and specific drugs in the future Firstly, the concept of a central common cell death pathway, originally derived from studies on the nematode, has been an outstanding productive paradigm in bringing together different strands of research. Secondly, truly striking links have been made between results obtained in the culture dish (or even cell-free systems) and the diseased human brain.
Da: Revaluation Books, Exeter, Regno Unito
EUR 234,87
Quantità: 2 disponibili
Aggiungi al carrelloHardcover. Condizione: Brand New. 1st edition. 154 pages. 9.75x6.50x0.50 inches. In Stock.
Da: Mispah books, Redhill, SURRE, Regno Unito
EUR 226,86
Quantità: 1 disponibili
Aggiungi al carrelloHardcover. Condizione: Like New. Like New. book.
Lingua: Inglese
Editore: Springer Berlin Heidelberg Sep 2001, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germania
EUR 160,49
Quantità: 2 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Four chapters represent the intense current effort to understand the way in which the mitochondrion controls the activation of the final stages of cell death. Another four articles attack the problem from the other side. How do specific insults in particular human or mouse neuro-degenerative diseases translate into mechanisms that will not only allow us to better understand what is happening in these patients but also, with luck, allow for development of more efficient and specific drugs in the future Firstly, the concept of a central common cell death pathway, originally derived from studies on the nematode, has been an outstanding productive paradigm in bringing together different strands of research. Secondly, truly striking links have been made between results obtained in the culture dish (or even cell-free systems) and the diseased human brain. 168 pp. Englisch.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: moluna, Greven, Germania
EUR 136,16
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Four chapters represent the intense current effort to understand the way in which the mitochondrion controls the activation of the final stages of cell death. Another four articles attack the problem from the other side. How do specific insults in particula.
Da: preigu, Osnabrück, Germania
EUR 141,20
Quantità: 5 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. Neuronal Death by Accident or by Design | C. E. Henderson (u. a.) | Buch | Einband - fest (Hardcover) | Englisch | 2001 | Springer-Verlag GmbH | EAN 9783540417774 | Verantwortliche Person für die EU: Springer Heidelberg, Tiergartenstr. 17, 69121 Heidelberg, buchhandel-buch[at]springer[dot]com | Anbieter: preigu Print on Demand.
Lingua: Inglese
Editore: Springer Berlin Heidelberg, Springer Berlin Heidelberg Sep 2001, 2001
ISBN 10: 354041777X ISBN 13: 9783540417774
Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germania
EUR 160,49
Quantità: 1 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Four chapters represent the intense current effort to understand the way in which the mitochondrion controls the activation of the final stages of cell death. Another four articles attack the problem from the other side. How do specific insults in particular human or mouse neuro-degenerative diseases translate into mechanisms that will not only allow us to better understand what is happening in these patients but also, with luck, allow for development of more efficient and specific drugs in the future Firstly, the concept of a central common cell death pathway, originally derived from studies on the nematode, has been an outstanding productive paradigm in bringing together different strands of research. Secondly, truly striking links have been made between results obtained in the culture dish (or even cell-free systems) and the diseased human brain.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 168 pp. Englisch.