Isbn: 9783659931048 - the role of hiv genetic pathway in apoptosis: hiv physiological role in cell death and survival of cells in the human immune system (7 risultati)

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    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing, 2016

      3659931047 / 9783659931048

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      Taschenbuch. Condizione: Neu. The Role of HIV Genetic Pathway in Apoptosis | HIV physiological role in cell death and survival of cells in the human immune system | Shahin Asadi | Taschenbuch | 164 S. | Englisch | 2016 | LAP LAMBERT Academic Publishing | EAN 9783659931048 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing, 2016

      3659931047 / 9783659931048

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      Da: Revaluation Books, Exeter, Regno UnitoRevaluation Books

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      Paperback. Condizione: Brand New. 164 pages. 8.66x5.91x0.37 inches. In Stock.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing, 2016

      3659931047 / 9783659931048

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      Da: Mispah books, Redhill, SURRE, Regno UnitoMispah books

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      paperback. Condizione: New. NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing Aug 2016, 2016

      3659931047 / 9783659931048

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      Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermaniaBuchWeltWeit Ludwig Meier e.K.

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      Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Today, HIV and AIDS is a global problem and epidemic disorders in the world. Every year the number of HIV infected individuals increases. Many studies have been done a cure for HIV, but the treatment is appropriate and decisive undiscovered. In this book, the physiological role of HIV in cell death was studied.Apoptosis of uninfected bystander cells is a key element of HIV pathogenesis and believed to be the driving force behind the selective depletion of CD4+ T cells leading to immunodeficiency. While several viral proteins have been implicated in this process the complex interaction between Env glycoprotein expressed on the surface of infected cells and the receptor and co-receptor expressing bystander cells has been proposed as a major mechanism. HIV-1 utilizes CD4 as the primary receptor for entry into cells; however, it is the viral co-receptor usage that greatly influences CD4 decline and progression to AIDS. This phenomenon is relatively simple for X4 viruses, which arise later during the course of the disease, are considered to be highly fusogenic, and cause a rapid CD4+ T cell decline. However, in contrast, R5 viruses in general have a greater transmissibility, are encount 164 pp. Englisch.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing, 2016

      3659931047 / 9783659931048

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      Da: moluna, Greven, Germaniamoluna

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      Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Asadi ShahinShahin Asadi,student of Molecular Genetics, Studied Molecular Biology at Tabriz Islamic Azad University Sciences.Bachelor of Moleculer and Cellular Biology-Genetics,Master of Moleculer Biology and Genetics Director of res.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing Aug 2016, 2016

      3659931047 / 9783659931048

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      Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germaniabuchversandmimpf2000

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      Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Today, HIV and AIDS is a global problem and epidemic disorders in the world. Every year the number of HIV infected individuals increases. Many studies have been done a cure for HIV, but the treatment is appropriate and decisive undiscovered. In this book, the physiological role of HIV in cell death was studied.Apoptosis of uninfected bystander cells is a key element of HIV pathogenesis and believed to be the driving force behind the selective depletion of CD4+ T cells leading to immunodeficiency. While several viral proteins have been implicated in this process the complex interaction between Env glycoprotein expressed on the surface of infected cells and the receptor and co-receptor expressing bystander cells has been proposed as a major mechanism. HIV-1 utilizes CD4 as the primary receptor for entry into cells; however, it is the viral co-receptor usage that greatly influences CD4 decline and progression to AIDS. This phenomenon is relatively simple for X4 viruses, which arise later during the course of the disease, are considered to be highly fusogenic, and cause a rapid CD4+ T cell decline. However, in contrast, R5 viruses in general have a greater transmissibility, are encountVDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 164 pp. Englisch.

    • Lingua: Inglese

      Editore: LAP LAMBERT Academic Publishing, 2016

      3659931047 / 9783659931048

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      Da: AHA-BUCH GmbH, Einbeck, GermaniaAHA-BUCH GmbH

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      Taschenbuch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Today, HIV and AIDS is a global problem and epidemic disorders in the world. Every year the number of HIV infected individuals increases. Many studies have been done a cure for HIV, but the treatment is appropriate and decisive undiscovered. In this book, the physiological role of HIV in cell death was studied.Apoptosis of uninfected bystander cells is a key element of HIV pathogenesis and believed to be the driving force behind the selective depletion of CD4+ T cells leading to immunodeficiency. While several viral proteins have been implicated in this process the complex interaction between Env glycoprotein expressed on the surface of infected cells and the receptor and co-receptor expressing bystander cells has been proposed as a major mechanism. HIV-1 utilizes CD4 as the primary receptor for entry into cells; however, it is the viral co-receptor usage that greatly influences CD4 decline and progression to AIDS. This phenomenon is relatively simple for X4 viruses, which arise later during the course of the disease, are considered to be highly fusogenic, and cause a rapid CD4+ T cell decline. However, in contrast, R5 viruses in general have a greater transmissibility, are encount.