Isbn: 9783838319636 - tackling a legendary scientific mystery: elucidation of the bacteriophage lambda t4rii exclusion phenotype (5 risultati)

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  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2010

    383831963X / 9783838319636

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    Da: preigu, Osnabrück, Germaniapreigu

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    Taschenbuch. Condizione: Neu. Tackling a Legendary Scientific Mystery | Elucidation of the Bacteriophage Lambda T4rII Exclusion Phenotype | Slavcev | Taschenbuch | 264 S. | Englisch | 2010 | LAP LAMBERT Academic Publishing | EAN 9783838319636 | Verantwortliche Person für die EU: BoD - Books on Demand, In de Tarpen 42, 22848 Norderstedt, info[at]bod[dot]de | Anbieter: preigu.…

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing Okt 2009, 2009

    383831963X / 9783838319636

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    Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermaniaBuchWeltWeit Ludwig Meier e.K.

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    Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critical role in our understanding of modern molecular genetics. Shockingly, since first exploited by Seymour Benzer in the mid 1950's, the mechanism underpinning the Rex phenotype remains unexplained. T4rII exclusion is encoded by the lambda genes rexA-rexB co-expressed with the lambda repressor from the prophage as the Pm-cI-rexA-rexB-timm operon. The onset of exclusion can result in the shutdown of cellular metabolism, preventing the replication of the super-infecting T4rII bacteriophage. In addition to reviewing the historic plight to characterize and resolve the Rex phenotype, we tackle this enigmatic phenomenon, adding important pieces to this puzzle and offer a working model for the mechanism of action. 264 pp. Englisch.…

  • Lingua: Inglese

    Editore: LAP Lambert Academic Publishing, 2009

    383831963X / 9783838319636

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    Da: moluna, Greven, Germaniamoluna

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    Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critica.…

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2010

    383831963X / 9783838319636

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    Da: AHA-BUCH GmbH, Einbeck, GermaniaAHA-BUCH GmbH

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    Taschenbuch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critical role in our understanding of modern molecular genetics. Shockingly, since first exploited by Seymour Benzer in the mid 1950's, the mechanism underpinning the Rex phenotype remains unexplained. T4rII exclusion is encoded by the lambda genes rexA-rexB co-expressed with the lambda repressor from the prophage as the Pm-cI-rexA-rexB-timm operon. The onset of exclusion can result in the shutdown of cellular metabolism, preventing the replication of the super-infecting T4rII bacteriophage. In addition to reviewing the historic plight to characterize and resolve the Rex phenotype, we tackle this enigmatic phenomenon, adding important pieces to this puzzle and offer a working model for the mechanism of action.…

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing Okt 2009, 2009

    383831963X / 9783838319636

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    Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germaniabuchversandmimpf2000

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    Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The T4rII exclusion (Rex) Phenotype is defined as the inability of rII mutants of bacteriophage T4 to generate plaques on a cell lawn of E. coli harboring a stably integrated lambda prophage. The selective exclusion of the rII mutants of T4 played a critical role in our understanding of modern molecular genetics. Shockingly, since first exploited by Seymour Benzer in the mid 1950''s, the mechanism underpinning the Rex phenotype remains unexplained. T4rII exclusion is encoded by the lambda genes rexA-rexB co-expressed with the lambda repressor from the prophage as the Pm-cI-rexA-rexB-timm operon. The onset of exclusion can result in the shutdown of cellular metabolism, preventing the replication of the super-infecting T4rII bacteriophage. In addition to reviewing the historic plight to characterize and resolve the Rex phenotype, we tackle this enigmatic phenomenon, adding important pieces to this puzzle and offer a working model for the mechanism of action.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 264 pp. Englisch.…