Isbn: 9786630098266 - emerging periodontal pathogens: evidence and clinical impact (9 risultati)

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  • Lingua: Inglese

    Editore: LAP Lambert Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: California Books, Miami, FL, U.S.A.California Books

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  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: PBShop.store US, Wood Dale, IL, U.S.A.PBShop.store US

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    PAP. Condizione: New. New Book. Shipped from UK. Established seller since 2000.

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: PBShop.store UK, Fairford, GLOS, Regno UnitoPBShop.store UK

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    PAP. Condizione: New. New Book. Shipped from UK. Established seller since 2000.

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: preigu, Osnabrück, Germaniapreigu

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    Taschenbuch. Condizione: Neu. Emerging Periodontal Pathogens: Evidence and clinical Impact | Megha N (u. a.) | Taschenbuch | Englisch | 2026 | LAP LAMBERT Academic Publishing | EAN 9786630098266 | Verantwortliche Person für die EU: SIA OmniScriptum Publishing, Brivibas Gatve 197, 1039 RIGA, LETTLAND, customerservice[at]vdm-vsg[dot]de | Anbieter: preigu.

  • Lingua: Inglese

    Editore: LAP Lambert Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: Grand Eagle Retail, Bensenville, IL, U.S.A.Grand Eagle Retail

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    Paperback. Condizione: new. Paperback. Periodontitis is now understood as a polymicrobial dysbiotic disease resulting from complex interactions between the subgingival microbiome and the host immune response. Earlier concepts focused on a limited number of pathogens, but the work of Sigmund Socransky established the importance of microbial complexes, particularly the red complex, Porphyromonas gingivalis, Tannerella forsythia, and Treponema denticola in disease progression.Advances in molecular techniques, especially next-generation sequencing, have expanded our understanding of the periodontal microbiome and led to the identification of newer periodontal pathogens. These include organisms such as Filifactor alocis, Enterococcus faecalis, Staphylococcus aureus, and Escherichia coli, which may act as pathobionts, contributing to disease under favorable environmental conditions. The newer periodontal pathogens have broadened the understanding of periodontal disease from a specific infection model to a complex, community-driven process. This evolving knowledge supports the development of targeted, personalized, and microbiome-based therapeutic approaches in contemporary periodontology. This item is printed on demand. Shipping may be from multiple locations in the US or from the UK, depending on stock availability.

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing Mai 2026, 2026

    6630098269 / 9786630098266

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    Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermaniaBuchWeltWeit Ludwig Meier e.K.

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    Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware 144 pp. Englisch.

  • Lingua: Inglese

    Editore: LAP Lambert Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: CitiRetail, Stevenage, Regno UnitoCitiRetail

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    Paperback. Condizione: new. Paperback. Periodontitis is now understood as a polymicrobial dysbiotic disease resulting from complex interactions between the subgingival microbiome and the host immune response. Earlier concepts focused on a limited number of pathogens, but the work of Sigmund Socransky established the importance of microbial complexes, particularly the red complex, Porphyromonas gingivalis, Tannerella forsythia, and Treponema denticola in disease progression.Advances in molecular techniques, especially next-generation sequencing, have expanded our understanding of the periodontal microbiome and led to the identification of newer periodontal pathogens. These include organisms such as Filifactor alocis, Enterococcus faecalis, Staphylococcus aureus, and Escherichia coli, which may act as pathobionts, contributing to disease under favorable environmental conditions. The newer periodontal pathogens have broadened the understanding of periodontal disease from a specific infection model to a complex, community-driven process. This evolving knowledge supports the development of targeted, personalized, and microbiome-based therapeutic approaches in contemporary periodontology. This item is printed on demand. Shipping may be from our UK warehouse or from our Australian or US warehouses, depending on stock availability.

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing Mai 2026, 2026

    6630098269 / 9786630098266

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    Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germaniabuchversandmimpf2000

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    Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware 144 pp. Englisch.

  • Lingua: Inglese

    Editore: LAP LAMBERT Academic Publishing, 2026

    6630098269 / 9786630098266

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    Da: AHA-BUCH GmbH, Einbeck, GermaniaAHA-BUCH GmbH

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    Taschenbuch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Periodontitis is now understood as a polymicrobial dysbiotic disease resulting from complex interactions between the subgingival microbiome and the host immune response. Earlier concepts focused on a limited number of pathogens, but the work of Sigmund Socransky established the importance of microbial complexes, particularly the red complex, Porphyromonas gingivalis, Tannerella forsythia, and Treponema denticola in disease progression.Advances in molecular techniques, especially next-generation sequencing, have expanded our understanding of the periodontal microbiome and led to the identification of newer periodontal pathogens. These include organisms such as Filifactor alocis, Enterococcus faecalis, Staphylococcus aureus, and Escherichia coli, which may act as pathobionts, contributing to disease under favorable environmental conditions. The newer periodontal pathogens have broadened the understanding of periodontal disease from a specific infection model to a complex, community-driven process. This evolving knowledge supports the development of targeted, personalized, and microbiome-based therapeutic approaches in contemporary periodontology.