Editore: Morgan & Claypool Publishers, 2018
ISBN 10: 1681734435 ISBN 13: 9781681734439
Lingua: Inglese
Da: suffolkbooks, Center moriches, NY, U.S.A.
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Editore: Springer International Publishing, Springer Nature Switzerland Nov 2018, 2018
ISBN 10: 3031012860 ISBN 13: 9783031012860
Lingua: Inglese
Da: buchversandmimpf2000, Emtmannsberg, BAYE, Germania
EUR 53,49
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Neuware -Atherosclerosis is a pathological condition of the arteries in which plaque buildup and stiffening (hardening) can lead to stroke, myocardial infarction (heart attacks), and even death. Cholesterol in the blood is a key marker for atherosclerosis, with two forms: (1) LDL - low density lipoproteins and (2) HDL - high density lipoproteins. Low LDL and high HDL concentrations are generally considered essential for limited atherosclerosis and good health.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 144 pp. Englisch.
Editore: Springer International Publishing, 2018
ISBN 10: 3031012860 ISBN 13: 9783031012860
Lingua: Inglese
Da: AHA-BUCH GmbH, Einbeck, Germania
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Atherosclerosis is a pathological condition of the arteries in which plaque buildup and stiffening (hardening) can lead to stroke, myocardial infarction (heart attacks), and even death. Cholesterol in the blood is a key marker for atherosclerosis, with two forms: (1) LDL - low density lipoproteins and (2) HDL - high density lipoproteins. Low LDL and high HDL concentrations are generally considered essential for limited atherosclerosis and good health.This book pertains to a mathematical model for the spatiotemporal distribution of LDL and HDL in the arterial endothelial inner layer (EIL, intima). The model consists of a system of six partial differential equations (PDEs) with the dependent variables1. ( , ): concentration of modified LDL2. ( , ): concentration of HDL3. ( , ): concentration of chemoattractants4. ( , ): concentration of ES cytokines5. ( , ): density of monocytes/macrophages6. ( , ): density of foam cellsand independent variables1. : distance from the inner arterial wall2. : timeThe focus of this book is a discussion of the methodology for placing the model on modest computers for study of the numerical solutions. The foam cell density ( , ) as a function of the bloodstream LDL and HDL concentrations is of particular interest as a precursor for arterial plaque formation and stiffening.The numerical algorithm for the solution of the model PDEs is the method of lines (MOL), a general procedure for the computer-based numerical solution of PDEs. The MOL coding (programming) is in R, a quality, open-source scientific computing system that is readily available from the Internet. The R routines for the PDE model are discussed in detail, and are available from a download link so that the reader/analyst/researcher can execute the model to duplicate the solutions reported in the book, then experiment with the model, for example, by changing the parameters (constants) and extending the model with additional equations.
Editore: Springer International Publishing Nov 2018, 2018
ISBN 10: 3031012860 ISBN 13: 9783031012860
Lingua: Inglese
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germania
EUR 53,49
Quantità: 2 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Atherosclerosis is a pathological condition of the arteries in which plaque buildup and stiffening (hardening) can lead to stroke, myocardial infarction (heart attacks), and even death. Cholesterol in the blood is a key marker for atherosclerosis, with two forms: (1) LDL - low density lipoproteins and (2) HDL - high density lipoproteins. Low LDL and high HDL concentrations are generally considered essential for limited atherosclerosis and good health.This book pertains to a mathematical model for the spatiotemporal distribution of LDL and HDL in the arterial endothelial inner layer (EIL, intima). The model consists of a system of six partial differential equations (PDEs) with the dependent variables1. ( , ): concentration of modified LDL2. ( , ): concentration of HDL3. ( , ): concentration of chemoattractants4. ( , ): concentration of ES cytokines5. ( , ): density of monocytes/macrophages6. ( , ): density of foam cellsand independent variables1. : distance from the inner arterial wall2. : timeThe focus of this book is a discussion of the methodology for placing the model on modest computers for study of the numerical solutions. The foam cell density ( , ) as a function of the bloodstream LDL and HDL concentrations is of particular interest as a precursor for arterial plaque formation and stiffening.The numerical algorithm for the solution of the model PDEs is the method of lines (MOL), a general procedure for the computer-based numerical solution of PDEs. The MOL coding (programming) is in R, a quality, open-source scientific computing system that is readily available from the Internet. The R routines for the PDE model are discussed in detail, and are available from a download link so that the reader/analyst/researcher can execute the model to duplicate the solutions reported in the book, then experiment with the model, for example, by changing the parameters (constants) and extending the model with additional equations. 144 pp. Englisch.
Da: Majestic Books, Hounslow, Regno Unito
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Aggiungi al carrelloCondizione: New. Print on Demand.
Da: Biblios, Frankfurt am main, HESSE, Germania
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Editore: Springer, Berlin|Springer International Publishing|Morgan & Claypool|Springer, 2018
ISBN 10: 3031012860 ISBN 13: 9783031012860
Lingua: Inglese
Da: moluna, Greven, Germania
EUR 47,23
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Aggiungi al carrelloCondizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Atherosclerosis is a pathological condition of the arteries in which plaque buildup and stiffening (hardening) can lead to stroke, myocardial infarction (heart attacks), and even death. Cholesterol in the blood is a key marker for atherosclerosis, with t.