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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Inhibin, Activin and Follistatin | Regulatory Functions in System and Cell Biology | Toshihiro Aono (u. a.) | Taschenbuch | xix | Englisch | 2012 | Springer | EAN 9781461273202 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Inhibin, activin, and follistatin were originally discovered as hormonal fac tors a decade ago. Subsequent studies revealed that they are widely distrib uted anatomically and have multiple biological functions in a wide range of cells and tissues. Since the last Symposium three years ago, there has been remarkable progress toward defining activin's roles in controlling hormone secretion, cell differentiation and proliferation, mesoderm induction in em bryos, nerve cell survival, and promotion of bone growth. Furthermore, the characterization of activin receptors as serine and threonine kinases and the identification of intracellular mediators such as the Smads have provided key elements for the eventual understanding of signaling mechanisms for activin and the other members of the TGF-beta superfamily. The unknown in this integrative field, however, remains far greater than the known. For ex ample, we are just beginning to exploit the recently improved activin, inhibin, and follistatin two-site assays for the evaluation of the physiological and pathophysiological functions of these proteins. Also, we have only a rudi mentary understanding of the mechanisms by which inhibin blocks activin, and many steps in the pathways linking the formation of ligand-receptor(s) complexes to transcriptional regulation are unclear. The aim of the Third Symposium was to bring together scientists from diverse fields to share technical and conceptual developments. The program was framed with the intention to include a wide range of timely research topics.
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Aggiungi al carrelloCondizione: New. This volume is the proceedings of the International Conference on Inhibin, Activin: Recent Advances and Future Views held in Tokushima, Japan from November 9-10, 1996. The Internationally recognized faculty present the latest research in the exploration of .
Da: AHA-BUCH GmbH, Einbeck, Germania
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Aggiungi al carrelloBuch. Condizione: Neu. Neuware - This volume is the proceedings of the International Conference on Inhibin, Activin: Recent Advances and Future Views held in Tokushima, Japan from November 9-10, 1996. The Internationally recognized faculty present the latest research in the exploration of inhibin, activin and follistatin mechanisms of action.
Data di pubblicazione: 1997
Da: Xerxes Fine and Rare Books and Documents, Glen Head, NY, U.S.A.
Condizione: Fine. NY 1997 1st Springer. ISBN 0-387-94969-0. 8vo., 333pp., 84 figures, index, printed hardcover. NEW. Small "final sale" stamp on front free blank o/w Fine.
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Aggiungi al carrelloCondizione: new. Questo è un articolo print on demand.
Lingua: Inglese
Editore: Springer New York Okt 2012, 2012
ISBN 10: 146127320X ISBN 13: 9781461273202
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germania
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Inhibin, activin, and follistatin were originally discovered as hormonal fac tors a decade ago. Subsequent studies revealed that they are widely distrib uted anatomically and have multiple biological functions in a wide range of cells and tissues. Since the last Symposium three years ago, there has been remarkable progress toward defining activin's roles in controlling hormone secretion, cell differentiation and proliferation, mesoderm induction in em bryos, nerve cell survival, and promotion of bone growth. Furthermore, the characterization of activin receptors as serine and threonine kinases and the identification of intracellular mediators such as the Smads have provided key elements for the eventual understanding of signaling mechanisms for activin and the other members of the TGF-beta superfamily. The unknown in this integrative field, however, remains far greater than the known. For ex ample, we are just beginning to exploit the recently improved activin, inhibin, and follistatin two-site assays for the evaluation of the physiological and pathophysiological functions of these proteins. Also, we have only a rudi mentary understanding of the mechanisms by which inhibin blocks activin, and many steps in the pathways linking the formation of ligand-receptor(s) complexes to transcriptional regulation are unclear. The aim of the Third Symposium was to bring together scientists from diverse fields to share technical and conceptual developments. The program was framed with the intention to include a wide range of timely research topics. 360 pp. Englisch.
Lingua: Inglese
Editore: Springer-Verlag New York Inc., 2012
ISBN 10: 146127320X ISBN 13: 9781461273202
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Aggiungi al carrelloCondizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. 1. Receptor Serine Kinases and Signaling by Activins and Inhibins.- 2. The Gene Expression of Activin Receptors in Rat Pituitary.- 3. Follistatin Physiology and Potential Mechanisms of Action in the Human.- 4. Inhibins, Activins, Follistatin, and GnRH: Regu.
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Aggiungi al carrelloCondizione: New. PRINT ON DEMAND pp. 360.
Lingua: Inglese
Editore: Springer, Springer Okt 2012, 2012
ISBN 10: 146127320X ISBN 13: 9781461273202
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Inhibin, activin, and follistatin were originally discovered as hormonal fac tors a decade ago. Subsequent studies revealed that they are widely distrib uted anatomically and have multiple biological functions in a wide range of cells and tissues. Since the last Symposium three years ago, there has been remarkable progress toward defining activin's roles in controlling hormone secretion, cell differentiation and proliferation, mesoderm induction in em bryos, nerve cell survival, and promotion of bone growth. Furthermore, the characterization of activin receptors as serine and threonine kinases and the identification of intracellular mediators such as the Smads have provided key elements for the eventual understanding of signaling mechanisms for activin and the other members of the TGF-beta superfamily. The unknown in this integrative field, however, remains far greater than the known. For ex ample, we are just beginning to exploit the recently improved activin, inhibin, and follistatin two-site assays for the evaluation of the physiological and pathophysiological functions of these proteins. Also, we have only a rudi mentary understanding of the mechanisms by which inhibin blocks activin, and many steps in the pathways linking the formation of ligand-receptor(s) complexes to transcriptional regulation are unclear. The aim of the Third Symposium was to bring together scientists from diverse fields to share technical and conceptual developments. The program was framed with the intention to include a wide range of timely research topics.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 360 pp. Englisch.
Lingua: Inglese
Editore: Springer-Verlag New York Inc., 1997
ISBN 10: 0387949690 ISBN 13: 9780387949697
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Aggiungi al carrelloCondizione: New. PRINT ON DEMAND pp. 333.