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Aggiungi al carrelloBrossura. Condizione: new. Roma, 2023; br., pp. 104, cm 16,5x24. Libro.
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ISBN 10: 3659268496 ISBN 13: 9783659268496
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Telomerase Activity in Normal and Neoplastic Lymph Nodes in the Dog | Telomerase, the Enzyme that Provides Neoplastic Cells of Immortality could be a Potential Target to Defeat Cancer | Fabio Valentini | Taschenbuch | 100 S. | Englisch | 2012 | LAP LAMBERT Academic Publishing | EAN 9783659268496 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu.
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Aggiungi al carrelloCondizione: Gut. Zustand: Gut | Seiten: 564 | Sprache: Italienisch | Produktart: Bücher | Keine Beschreibung verfügbar.
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -One of the several differences between a normal and a neoplastic cell is that there is a physiological limit to the number of possible cell replications in the former, whereas it appears to be unlimited in the latter. For this reason, cancer cells are thought to undergo a phenomenon of immortalization. The chromosome extremities contain structurally defined elements called telomeres. In normal somatic cells, each mitotic cycle leads to a reduction of telomere length until a critical point is reached. Telomere erosion to this critical length presents a signal for the cell to arrest further divisions and to undergo cellular senescence or to activate apoptosis. Telomeres that avoid critical shortening could, theoretically, replicate endlessly, thereby immortalizing the cell. One mechanism to restore telomere length is the activation of a specific reverse transcriptase called telomerase. Up-regulation of the telomerase enzyme provides immortalizing capacity to neoplastic cells. 100 pp. Englisch.
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Da: moluna, Greven, Germania
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Aggiungi al carrelloCondizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Valentini FabioDr Valentini graduated at the University of Bologna (Italy) in 1999. One year later he moved to the University of Illinois pursuing a 2-year MS and clinical oncology internship. In 2010 he started his residency in med.
Lingua: Inglese
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ISBN 10: 3659268496 ISBN 13: 9783659268496
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -One of the several differences between a normal and a neoplastic cell is that there is a physiological limit to the number of possible cell replications in the former, whereas it appears to be unlimited in the latter. For this reason, cancer cells are thought to undergo a phenomenon of immortalization. The chromosome extremities contain structurally defined elements called telomeres. In normal somatic cells, each mitotic cycle leads to a reduction of telomere length until a critical point is reached. Telomere erosion to this critical length presents a signal for the cell to arrest further divisions and to undergo cellular senescence or to activate apoptosis. Telomeres that avoid critical shortening could, theoretically, replicate endlessly, thereby immortalizing the cell. One mechanism to restore telomere length is the activation of a specific reverse transcriptase called telomerase. Up-regulation of the telomerase enzyme provides immortalizing capacity to neoplastic cells.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 100 pp. Englisch.
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ISBN 10: 3659268496 ISBN 13: 9783659268496
Da: AHA-BUCH GmbH, Einbeck, Germania
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - One of the several differences between a normal and a neoplastic cell is that there is a physiological limit to the number of possible cell replications in the former, whereas it appears to be unlimited in the latter. For this reason, cancer cells are thought to undergo a phenomenon of immortalization. The chromosome extremities contain structurally defined elements called telomeres. In normal somatic cells, each mitotic cycle leads to a reduction of telomere length until a critical point is reached. Telomere erosion to this critical length presents a signal for the cell to arrest further divisions and to undergo cellular senescence or to activate apoptosis. Telomeres that avoid critical shortening could, theoretically, replicate endlessly, thereby immortalizing the cell. One mechanism to restore telomere length is the activation of a specific reverse transcriptase called telomerase. Up-regulation of the telomerase enzyme provides immortalizing capacity to neoplastic cells.
ISBN 10: 6525147611 ISBN 13: 9786525147611
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