Da: Anybook.com, Lincoln, Regno Unito
EUR 24,61
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Fair. Volume 7. This is an ex-library book and may have the usual library/used-book markings inside.This book has soft covers. In fair condition, suitable as a study copy. Please note the Image in this listing is a stock photo and may not match the covers of the actual item,650grams, ISBN:9789048181278.
Da: Anybook.com, Lincoln, Regno Unito
EUR 29,10
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Poor. Volume 7. This is an ex-library book and may have the usual library/used-book markings inside.This book has soft covers. In poor condition, suitable as a reading copy. Please note the Image in this listing is a stock photo and may not match the covers of the actual item,600grams, ISBN:9789048181278.
Da: Anybook.com, Lincoln, Regno Unito
EUR 30,93
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Fair. Volume 7. This is an ex-library book and may have the usual library/used-book markings inside.This book has soft covers. In fair condition, suitable as a study copy. Please note the Image in this listing is a stock photo and may not match the covers of the actual item,650grams, ISBN:9789048181278.
Da: Anybook.com, Lincoln, Regno Unito
EUR 34,21
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Fair. Volume 7. This is an ex-library book and may have the usual library/used-book markings inside.This book has hardback covers. In fair condition, suitable as a study copy. No dust jacket. Please note the Image in this listing is a stock photo and may not match the covers of the actual item,650grams, ISBN:9781402094330.
Da: Anybook.com, Lincoln, Regno Unito
EUR 35,75
Quantità: 1 disponibili
Aggiungi al carrelloCondizione: Good. Volume 7. This is an ex-library book and may have the usual library/used-book markings inside.This book has hardback covers. In good all round condition. No dust jacket. Please note the Image in this listing is a stock photo and may not match the covers of the actual item,650grams, ISBN:9781402094330.
Da: Oriental Research Partners, Newtonville, MA, U.S.A.
Hardcover. Condizione: As New. Budapest 1988/, pp. 180. hc/as new. Intriguing photos of Hungarian immigration to America and arriving at Ellis Island; types of immigrants; early settlements/businesses in the new USA. In Hungarian. ISBN 9632820150.
Editore: Sopron, 1747. (12), 401, (7) pp. Text in Latin., 1747
Da: Antiquariaat Hortus Conclusus, Bergambacht, Paesi Bassi
EUR 45,00
Quantità: 1 disponibili
Aggiungi al carrelloContemporary full leather binding, but binding rather worn (esp. on spine). Frontispice engraving glued to verso front side, frontispice and first few leaves with small wormhole, some minor browning, else a very acceptable / good and complete copy. Please see description or ask for photos.
Da: Ria Christie Collections, Uxbridge, Regno Unito
EUR 153,39
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. In.
Da: Ria Christie Collections, Uxbridge, Regno Unito
EUR 165,70
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. In.
Da: Ria Christie Collections, Uxbridge, Regno Unito
EUR 165,70
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New. In.
Da: GreatBookPricesUK, Woodford Green, Regno Unito
EUR 165,68
Quantità: 3 disponibili
Aggiungi al carrelloCondizione: New.
Da: GreatBookPrices, Columbia, MD, U.S.A.
EUR 182,68
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: New.
EUR 140,10
Quantità: 5 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. Protein folding and misfolding: neurodegenerative diseases | Judit Ovádi (u. a.) | Taschenbuch | xiv | Englisch | 2010 | Springer | EAN 9789048181278 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
Da: Books Puddle, New York, NY, U.S.A.
Condizione: New. 1st ed. 2022 edition NO-PA16APR2015-KAP.
Condizione: New. pp. 292.
Condizione: New. pp. 292.
Da: Books Puddle, New York, NY, U.S.A.
Condizione: New. 1st ed. 2022 edition NO-PA16APR2015-KAP.
Lingua: Inglese
Editore: Springer Netherlands, Springer Netherlands, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Da: AHA-BUCH GmbH, Einbeck, Germania
EUR 164,49
Quantità: 1 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
EUR 168,73
Quantità: 1 disponibili
Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
Da: Revaluation Books, Exeter, Regno Unito
EUR 237,43
Quantità: 2 disponibili
Aggiungi al carrelloPaperback. Condizione: Brand New. 277 pages. 9.25x6.10x0.59 inches. In Stock.
Da: Revaluation Books, Exeter, Regno Unito
EUR 237,63
Quantità: 2 disponibili
Aggiungi al carrelloPaperback. Condizione: Brand New. 288 pages. 9.25x6.10x0.66 inches. In Stock.
Editore: R?zsav?lgyi, Pest, 1862
Da: Adam Bosze Music Antiquarian, Budapest, BUDAP, Ungeria
Spartito
EUR 25,00
Quantità: 1 disponibili
Aggiungi al carrelloSoftcover. Condizione: Used. Orosz, Ferenc: Eredeti magyar dallok, 1862, R?zsav?lgyi ?s t?rsa, 5 oldal.
Da: Revaluation Books, Exeter, Regno Unito
EUR 239,88
Quantità: 2 disponibili
Aggiungi al carrelloHardcover. Condizione: Brand New. 277 pages. 9.25x6.10x0.71 inches. In Stock.
Da: Revaluation Books, Exeter, Regno Unito
EUR 240,77
Quantità: 2 disponibili
Aggiungi al carrelloHardcover. Condizione: Brand New. 1st edition. 274 pages. 9.40x6.30x1.00 inches. In Stock.
Da: GreatBookPricesUK, Woodford Green, Regno Unito
EUR 266,86
Quantità: 3 disponibili
Aggiungi al carrelloCondizione: As New. Unread book in perfect condition.
Da: Mispah books, Redhill, SURRE, Regno Unito
EUR 257,25
Quantità: 1 disponibili
Aggiungi al carrelloHardcover. Condizione: Like New. Like New. book.
Da: GreatBookPrices, Columbia, MD, U.S.A.
EUR 292,63
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: As New. Unread book in perfect condition.
Da: Brook Bookstore On Demand, Napoli, NA, Italia
EUR 126,26
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: new. Questo è un articolo print on demand.
Da: Brook Bookstore On Demand, Napoli, NA, Italia
EUR 126,26
Quantità: Più di 20 disponibili
Aggiungi al carrelloCondizione: new. Questo è un articolo print on demand.
Lingua: Inglese
Editore: Springer Netherlands Jan 2009, 2009
ISBN 10: 1402094337 ISBN 13: 9781402094330
Da: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germania
EUR 160,49
Quantità: 2 disponibili
Aggiungi al carrelloBuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably. 296 pp. Englisch.