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Da: Basi6 International, Irving, TX, U.S.A.
Condizione: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.
Da: Romtrade Corp., STERLING HEIGHTS, MI, U.S.A.
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Aggiungi al carrelloGebunden. Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Covers the understanding of the role of activation-induced cytidine deaminase (AID) in the generation of antibody response to antigenic challenge.InhaltsverzeichnisSwitch Regions, Chromatin Accessibility and AID Targeting Cis-Regula.
Da: preigu, Osnabrück, Germania
EUR 93,50
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Aggiungi al carrelloBuch. Condizione: Neu. DNA DEAMINATION & THE IMMUNE SYSTEM (V3) | Fugmann Sebastian | Buch | Gebunden | Englisch | 2010 | ICP | EAN 9781848165922 | Verantwortliche Person für die EU: Libri GmbH, Europaallee 1, 36244 Bad Hersfeld, gpsr[at]libri[dot]de | Anbieter: preigu Print on Demand.
Da: AHA-BUCH GmbH, Einbeck, Germania
EUR 111,68
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Aggiungi al carrelloBuch. Condizione: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - This monograph covers current understanding of the role of activation induced cytidine deaminase (AID) in the generation of antibody response to antigenic challenge. Since the discovery of AID and the genetic demonstration of its role in somatic hypermutation and class switch recombination of antibody genes, much has been learned about the biochemistry of this enzyme. However, some key questions (How does this enzyme get to the antibody locus leaving the rest of the genome intact Why are DNA repair pathways which normally repair deamination events co-opted into actually fixing mutations into the genome ) remain hotly contested. These questions will be addressed in this monograph from various perspectives. Being leading experts in their respective fields, the contributors of this highly valued monograph summarize current research in the field of AID and put forth hypotheses in order to provide a platform for future experiments.