Koji shimoji (26 risultati)

Hardcover. Condizione: Très bon. Ancien livre de bibliothèque. Edition 1996. Ammareal reverse jusqu'à 15% du prix net de cet article à des organisations caritatives. ENGLISH DESCRIPTION Book Condition: Used, Very good. Former library book. Edition 1996. Ammareal gives back up to 15% of this item's net price to charity organizations.…

Hardcover. Condizione: Très bon. Ancien livre de bibliothèque. Edition 1996. Ammareal reverse jusqu'à 15% du prix net de cet article à des organisations caritatives. ENGLISH DESCRIPTION Book Condition: Used, Very good. Former library book. Edition 1996. Ammareal gives back up to 15% of this item's net price to charity organizations.…

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Condizione: Used. pp. viii + 164 Index.

Condizione: Used. pp. viii + 164 Illus. (Some Col.).

Condizione: Used. pp. viii + 164.

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Condizione: New. In English.

Condizione: New. In English.

Condizione: New. pp. 176.

Paperback. Condizione: Brand New. 176 pages. 9.25x6.10x0.42 inches. In Stock.

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Taschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology.…

Buch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology.…

1. Auflage.. with 52 Figures Including 2 in Color, 164 Seiten, Zustand: erstklassiges, neuwertiges Exemplar aus der Bibliothek eines namhaften Neurologen! Sprache: Englisch Gewicht in Gramm: 800 24,0 x 16,0 cm, gebundene Ausgabe, Hardcover/Pappeinband.

Condizione: new. Questo è un articolo print on demand.

Buch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology. 176 pp. Englisch.…

Taschenbuch. Condizione: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology. 176 pp. Englisch. …

Gebunden. Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Functional Proteins in the Presynaptic Terminal.- Molecular Structure and Physiological Function of the Glutamate Receptor Channel.- Metabotropic Glutamate Responses and the Intracellular Mechanisms.- Long-Term Potentiation and Drugs.- Analysis of Distribut.…

Kartoniert / Broschiert. Condizione: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Functional Proteins in the Presynaptic Terminal.- Molecular Structure and Physiological Function of the Glutamate Receptor Channel.- Metabotropic Glutamate Responses and the Intracellular Mechanisms.- Long-Term Potentiation and Drugs.- Analysis of Distribut.…

Condizione: New. Print on Demand pp. 176 52 Figures (2 Col.), 49:B&W 6.14 x 9.21 in or 234 x 156 mm (Royal 8vo) Perfect Bound on White w/Gloss Lam.

Condizione: New. PRINT ON DEMAND pp. 176.

Taschenbuch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 176 pp. Englisch.…

Buch. Condizione: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Cerebral hypoxia/ischemia is a common cause of neurologic dysfunction following cardiopulmonary resuscitation. With the advent of aggressive resuscitatiQ,il ;m.ethods to treat cardiopulmonary arrest, more patients are surviving to live with posthypoxic/ postischemic neurologic syndromes. Thus, there is an urgent need to understand the basic aspects of the problem. This monograph consists of reviews from :s'c5ientists using advanced techniques for basic research into the molecular neurob~ology of the mechanisms and effects of cerebral hypoXia/ischemia, and from cliniciahs ~ctively involved in managing patients with these syndromes. The first five chapters contain several basic topics of neurobiology to help under stand brain function, followed by five chapters that deal with the mechanisms of cerebral hypoxia/ischemia and potential protective measures against it. The first chapter describes functional proteins in the presynaptic terminal which regulate transmitter release. This is followed by a chapter demonstrating the molecu lar structure and physiological function of the glutamate receptor channel, which may determine the rate and quantity of calcium ion influx into the nerve cell. The third chapter surveys the metabolic glutamate responses and the intracellular mechanisms believed to play an important role in pathogenesis of hypoxic/ischemic cell injury. The fourth chapter deals with long-term potentiation and drugs, the basis for under standing memory disturbance caused by cerebral hypoxia/ischemia. The fifth chapter describes distributions of nitric oxide synthase mRNAs in the brain, currently one of the most advanced fields in neurobiology.Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 176 pp. Englisch.…