Lingua: Inglese
Editore: Martinus Nijhoff Publishers, 1987
ISBN 10: 0898388708 ISBN 13: 9780898388701
Da: P.C. Schmidt, Bookseller, Kettering, OH, U.S.A.
Prima edizione
Hard Cover. Condizione: Very Good. First Edition. a very good hardcover; an ex-library copy with usual markings; internally pages bright and clean; ---------- SATISFACTION GUARANTEED --------- FAST, COURTEOUS SERVICE ------ ALL ORDERS SHIPPED WITH DELIVERY CONFIRMATION; Size: 6 x 9". Ex-Library.
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Aggiungi al carrelloPaperback. Condizione: New. Softcover reprint of the original 1st ed. 1988.
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Aggiungi al carrelloPaperback. Condizione: New.
Da: Books Puddle, New York, NY, U.S.A.
Condizione: New. pp. 164 Index.
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Aggiungi al carrelloPaperback. Condizione: Brand New. 164 pages. 9.25x6.10x0.37 inches. In Stock.
Da: Books Puddle, New York, NY, U.S.A.
Condizione: New. pp. 304.
Lingua: Inglese
Editore: Martinus Nijoff Publishers, 2013
ISBN 10: 9400967616 ISBN 13: 9789400967618
Da: Revaluation Books, Exeter, Regno Unito
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Aggiungi al carrelloPaperback. Condizione: Brand New. 304 pages. 9.25x6.10x0.69 inches. In Stock.
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Aggiungi al carrelloCondizione: New. H. E. D. J. TER KEURS & M. I. M. NOBLE The Starling s Law of the Heart and The Frank-Starling Mechanism have long been the cornerstone of cardiac mechanical physiology. It is often forgotten that Frank and Starling carried out fundamentally different ex.
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - H. E. D. J. TER KEURS & M. I. M. NOBLE The 'Starling's Law of the Heart' and 'The Frank-Starling Mechanism' have long been the cornerstone of cardiac mechanical physiology. It is often forgotten that Frank and Starling carried out fundamentally different exper iments. Frankl measured the isovolumic pressure developed by frog heart at different volumes. He therefore discovered the pressure-volume-volume rela tionship which depends directly on the force-length relationship of the 2 sarcomeres. Starling ,3 studied cardiac shortening as manifest by cardiac output and its relationship to end-diastolic conditions as manifest by right atrial pressure. Thus he was studying the ability of cardiac muscle to shorten more at a given load from a greater initial length. Starling in the promulga 4 tions of his law implied a common mechanism for these two phenomena and spoke of the 'energy liberated' being a function of initial muscle fiber length. However, there has been much confusion about the interrelationship between the two different aspects studied by Frank and Starling. The 1960s saw the era of isolated cardiac muscle mechanics, beginning with 5 the paper of Abbott and Mommaerts. Whole muscle length-tension relations were equated with sarcomere-length-tension relations by fixation of muscle at a particular point on the curve and determination of sarcomere length by electronmicroscopy.
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Cardiac Left Ventricular Hypertrophy | Henk Keurs (u. a.) | Taschenbuch | Developments in Cardiovascular Medicine | 300 S. | Englisch | 2011 | Springer | EAN 9789400967618 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Starling's Law of The Heart Revisited | Henk Keurs (u. a.) | Taschenbuch | Developments in Cardiovascular Medicine | 168 S. | Englisch | 2011 | Springer | EAN 9789401070843 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
Da: AHA-BUCH GmbH, Einbeck, Germania
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Aggiungi al carrelloTaschenbuch. Condizione: Neu. Druck auf Anfrage Neuware - Printed after ordering - Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria.
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Aggiungi al carrelloPaperback. Condizione: New. Softcover reprint of the original 1st ed. 1988.
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Da: Ria Christie Collections, Uxbridge, Regno Unito
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